sexta-feira, 19 de agosto de 2011

Será que a Maconha não é Remédio, Doutor Laranjeiras?! [Semente, né?! #129]

Todo mundo já conhece o Dr. Laranjeiras e o seu Dossiê. Ter essa atenção toda não é atoa, ele é quem mais prega o ódio ao usuário, as calúnias sobre a maconha, ele representa o status quo da proibição no seu lado mais encrustado e isso é sinônimo de orgulho que ele adora esbanjar. Uma das maiores falsas verdades do Doutor é que a maconha não tem nenhum potencial médico. A galera do blog preparou não só um post, mas um vídeo nota 10 para ilustrar esse Semente, né?!

 

20 comentários:

  1. isso ae eh do cortina de fumaça nee ! mais mesmo assim é muito legal ja passa a mensagem ! Boaa =)

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  2. http://www.youtube.com/watch?v=1DeAymh7Rhc&NR=1

    A juíza Maria Lúcia calma é linda me arrepiou. Humanismo, solidariedade. Deixem-nos plantar em paz.

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  3. Pô, eu ia escrever uma coisa sobre a juíza, aí mudei de idéia, mas esqueci de apagar uma parte, aí saiu esse troço meio sem sentido aí em cima. Mas o que ela diz, como diz, me arrepiou. É isso.

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  4. A proibição ajudou em que famílias com usuários 'crônicos', digamos assim, de drogas pesadas? Impediu que alguém comprasse? Só fez acrescentar outro problema. Então pra familiares só digo que procure outro pra tentar espancar, pra tentar descarregar a revolta. Que tal culpar governos, o poder, o estatismo? São os principais interessados em que sejamos bodes expiatórios. Ou, em muitos casos, culpados são as próprias famílias. Como em filmes policiais e na vida real, geralmente quem aponta dedo procurando culpado é o autor do crime.

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  5. Sou um dos que escreve pro blog e gostariamos de informar que o vídeo não foi feito por nós, mas sim o encontramos na internet.

    Valeu pelo apoio galera, vamos acabar com as mentiras desse doutor de "verdade"

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  6. é importante a simpatia de todos nesta luta do direito contra a violência. o brasil precisa apontar rumo à civilidade.

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  7. a intervenção estatal deve ser mínima. o estado nasceu para resolver problemas, e não para criar conflitos contra seus cidadãos pacíficos. as autoridades têm o dever de conduzir e de melhor administrar a justiça e os destinos da nação, não podendo aliar-se a forças visivelmente beligerantes e reacionárias.

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  8. me sinto envergonhado por ser natural de araçatuba e ouvir uma monstruosidade desta. tsc tsc tsc; bóra fumar um.

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  9. EXCELENTE!!!!!! TEMOS QUE MOSTRAR CADA VEZ MAIS AS CONTRADIÇÕES DOS PROIBICIONISTAS, ESFREGAR NA CARA DELES NOSSO EMBASSAMENTO!!! MANDOU MUITO BEM HEMPADÃO, CONTINUEM ASSIM!!!! SÓ ASSIM VAMOS DESMASCARAR ESSES PROIBICIONISTAS HIPOCRITAS!!!! LEGALIZE BRASIL!!!!!!

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  10. E os faladores que pelo mundo existe ainda pensam que a maconha é igual a crack ¬¬

    não falo nada pra quem pensa que a cannabis é a porta de entrada pra outras drogas mais pesadas.

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  11. Mais...

    ITALY / ITÁLIA
    ‪http://www.ncbi.nlm.nih.gov/pubmed/12052046
    Endocannabinoids in the immune system and cancer.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/19189054
    A metabolically stable analogue of anandamide, Met-F-AEA, inhibits human thyroid carcinoma cell lines by activation of apoptosis.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/18354058
    ‪Plant-derived cannabinoids modulate the activity of transient receptor potential channels of ankyrin type-1 and melastatin type-8.

    http://www.ncbi.nlm.nih.gov/pubmed/19047095
    Cannabinoid receptor activation induces apoptosis through tumor necrosis factor alpha-mediated ceramide de novo synthesis in colon cancer cells.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/10913156
    Anandamide induces apoptosis in human cells via vanilloid receptors. Evidence for a protective role of cannabinoid receptors.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/9653194
    The endogenous cannabinoid anandamide inhibits human breast cancer cell proliferation.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/18088200
    Cannabinoids as potential new therapy for the treatment of gliomas.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/16909207
    The non-psychoactive cannabidiol triggers caspase activation and oxidative stress in human glioma cells.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/17342320
    ‪Endocannabinoids as emerging suppressors of angiogenesis and tumor invasion (review).

    http://www.ncbi.nlm.nih.gov/pubmed/19059457
    Apoptosis induced in HepG2 cells by the synthetic cannabinoid WIN: involvement of the transcription factor PPARgamma.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/12723496
    The endocannabinoid system as a target for the development of new drugs for cancer therapy.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/19442536
    Cannabinoids in intestinal inflammation and cancer.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/16728591
    Antitumor activity of plant cannabinoids with emphasis on the effect of cannabidiol on human breast carcinoma.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/19539619
    The CB1/CB2 receptor agonist WIN-55,212-2 reduces viability of human Kaposi's sarcoma cells in vitro.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/16500647
    Cannabinoid derivatives induce cell death in pancreatic MIA PaCa-2 cells via a receptor-independent mechanism.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/19189659
    The endocannabinoid anandamide neither impairs in vitro T-cell function nor induces regulatory T-cell generation.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/14617682
    Antitumor effects of cannabidiol, a nonpsychoactive cannabinoid, on human glioma cell lines.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/18938775
    Estrogenic induction of cannabinoid CB1 receptor in human colon cancer cell lines.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/11106791
    Endocannabinoids and fatty acid amides in cancer, inflammation and related disorders.
    JAPAN / JAPÃO
    ‪http://www.ncbi.nlm.nih.gov/pubmed/19394652
    Pharmacological synergism between cannabinoids and paclitaxel in gastric cancer cell lines.

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  12. mais e mais :)

    KOREA / KORÉIA
    ‪http://www.ncbi.nlm.nih.gov/pubmed/20336665
    Effect of a synthetic cannabinoid agonist on the proliferation and invasion of gastric cancer cells.


    ‪NEW ZEALAND / NOVA ZELÂNDIA
    ‪http://www.ncbi.nlm.nih.gov/pubmed/19442435
    Cannabinoids in the treatment of cancer.

    ‪POLAND / POLÔNIA
    ‪http://www.ncbi.nlm.nih.gov/pubmed/15451022
    Cannabinoids down-regulate PI3K/Akt and Erk signalling pathways and activate proapoptotic function of Bad protein.


    ‪SAUDI ARABIA / ARÁBIA SAUDITA
    http://www.ncbi.nlm.nih.gov/pubmed/18197164
    Cannabinoid 2 receptor induction by IL-12 and its potential as a therapeutic target for the treatment of anaplastic thyroid carcinoma.


    ‪SLOVAKIA / SLOVÁQUIA
    ‪http://www.ncbi.nlm.nih.gov/pubmed/16835997
    Different views on the association between cannabinoids and cancer


    ‪SPAIN / ESPANHA
    ‪http://www.ncbi.nlm.nih.gov/pubmed/11903061
    De novo-synthesized ceramide is involved in cannabinoid-induced apoptosis.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/17675107
    Down-regulation of tissue inhibitor of metalloproteinases-1 in gliomas: a new marker of cannabinoid antitumoral activity?

    ‪http://www.ncbi.nlm.nih.gov/pubmed/17202146
    Cannabinoids induce glioma stem-like cell differentiation and inhibit gliomagenesis.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/19425170
    Cannabinoid action induces autophagy-mediated cell death through stimulation of ER stress in human glioma cells

    ‪http://www.ncbi.nlm.nih.gov/pubmed/18454173
    JunD is involved in the antiproliferative effect of Delta9-tetrahydrocannabinol on human breast cancer cells.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/17065222
    Cannabinoid receptors as novel targets for the treatment of melanoma.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/10700234
    Anti-tumoral action of cannabinoids: involvement of sustained ceramide accumulation and extracellular signal-regulated kinase activation.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/16787257
    Endocannabinoids: a new family of lipid mediators involved in the regulation of neural cell development.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/15958274
    Cannabinoids and ceramide: two lipids acting hand-by-hand.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/16139274
    p38 MAPK is involved in CB2 receptor-induced apoptosis of human leukaemia cells.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/16624285
    The CB2 cannabinoid receptor signals apoptosis via ceramide-dependent activation of the mitochondrial intrinsic pathway.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/16616335
    The stress-regulated protein p8 mediates cannabinoid-induced apoptosis of tumor cells.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/11269508
    Control of the cell survival/death decision by cannabinoids.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/19690545
    Inhibition of human tumour prostate PC-3 cell growth by cannabinoids R(+)-Methanandamide and JWH-015: involvement of CB2.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/12511587
    Inhibition of skin tumor growth and angiogenesis in vivo by activation of cannabinoid receptors.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/20307616
    Opposite changes in cannabinoid CB1 and CB2 receptor expression in human gliomas.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/16818634
    Delta9-tetrahydrocannabinol inhibits cell cycle progression in human breast cancer cells through Cdc2 regulation.

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  13. http://www.ncbi.nlm.nih.gov/pubmed/17952650
    Cannabinoids and gliomas.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/16818650
    Cannabinoids induce apoptosis of pancreatic tumor cells via endoplasmic reticulum stress-related genes.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/16596790
    Effects on cell viability.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/15638794
    Involvement of cannabinoids in cellular proliferation.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/15275820
    Hypothesis: cannabinoid therapy for the treatment of gliomas?

    ‪http://www.ncbi.nlm.nih.gov/pubmed/12133838
    Cannabinoids protect astrocytes from ceramide-induced apoptosis through the phosphatidylinositol 3-kinase/protein kinase B pathway.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/18339876
    Cannabinoids inhibit glioma cell invasion by down-regulating matrix metalloproteinase-2 expression.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/9771884
    Delta9-tetrahydrocannabinol induces apoptosis in C6 glioma cells.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/10570948
    Delta9-tetrahydrocannabinol induces apoptosis in human prostate PC-3 cells via a receptor-independent mechanism.


    ‪http://www.ncbi.nlm.nih.gov/pubmed/12182964
    Cannabinoids and cell fate.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/19229996
    Amphiregulin is a factor for resistance of glioma cells to cannabinoid-induced apoptosis.


    ‪SWEDEN / SUÉCIA‬
    ‪http://www.ncbi.nlm.nih.gov/pubmed/19609004
    Potentiation of cannabinoid-induced cytotoxicity in mantle cell lymphoma through modulation of ceramide metabolism.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/16337199
    Cannabinoid receptor ligands mediate growth inhibition and cell death in mantle cell lymphoma.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/16936228
    Cannabinoid receptor-mediated apoptosis induced by R(+)-methanandamide and Win55,212-2 is associated with ceramide accumulation and p38 activation in mantle cell lymphoma.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/18546271
    Expression of cannabinoid receptors type 1 and type 2 in non-Hodgkin lymphoma: growth inhibition by receptor activation.


    ‪SWITZERLAND / SUÍÇA
    ‪http://www.ncbi.nlm.nih.gov/pubmed/15453094
    Arachidonylethanolamide induces apoptosis of human glioma cells through vanilloid receptor-1.

    ResponderExcluir
  14. http://www.ncbi.nlm.nih.gov/pubmed/19589225
    Cannabinoid receptor ligands as potential anticancer agents--high hopes for new therapies?

    ‪http://www.ncbi.nlm.nih.gov/pubmed/15047233
    Arachidonyl ethanolamide induces apoptosis of uterine cervix cancer cells via aberrantly expressed vanilloid receptor-1.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/19509271
    Cannabinoid receptor 1 is a potential drug target for treatment of translocation-positive rhabdomyosarcoma.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/19480992
    Predominant CB2 receptor expression in endothelial cells of glioblastoma in humans.

    TAIWAN / TAIWAN
    ‪http://www.ncbi.nlm.nih.gov/pubmed/18387516
    A comparative study on cannabidiol-induced apoptosis in murine thymocytes and EL-4 thymoma cells.


    ‪THAILAND / TAILÂNDIA
    ‪http://www.ncbi.nlm.nih.gov/pubmed/19916793
    The dual effects of delta(9)-tetrahydrocannabinol on cholangiocarcinoma cells: anti-invasion activity at low concentration and apoptosis induction at high concentration.

    ‪UKRAINE / UCRÂNIA
    ‪http://www.ncbi.nlm.nih.gov/pubmed/18438336
    Antineoplastic and apoptotic effects of cannabinoids. N-acylethanolamines: protectors or killers?


    ‪UNITED KINGDOM / INGLATERRA
    ‪http://www.ncbi.nlm.nih.gov/pubmed/15454482
    Cannabis-induced cytotoxicity in leukemic cell lines: the role of the cannabinoid receptors and the MAPK pathway.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/17583570‬
    The cannabinoid delta(9)-tetrahydrocannabinol inhibits RAS-MAPK and PI3K-AKT survival signalling and induces BAD-mediated apoptosis in colorectal cancer cells.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/17931597
    Cannabinoid receptor agonists are mitochondrial inhibitors: a unified hypothesis of how cannabinoids modulate mitochondrial function and induce cell death.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/18615640
    High concentrations of cannabinoids activate apoptosis in human U373MG glioma cells.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/14640910
    Cannabinoid receptor systems: therapeutic targets for tumour intervention.

    ResponderExcluir
  15. ‪UNITED STATES OF AMERICA / ESTADOS UNIDOS DA AMERICA‬
    ‪http://www.ncbi.nlm.nih.gov/pubmed/20191092
    Cannabinoids as novel anti-inflammatory drugs.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/18025276
    Cannabidiol as a novel inhibitor of Id-1 gene expression in aggressive breast cancer cells.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/616322
    In vivo effects of cannabinoids on macromolecular biosynthesis in Lewis lung carcinomas.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/15753356
    Cannabinoid receptor as a novel target for the treatment of prostate cancer.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/12091357
    Targeting CB2 cannabinoid receptors as a novel therapy to treat malignant lymphoblastic disease.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/18199524
    Cannabinoids for cancer treatment: progress and promise.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/19887554
    Synthetic cannabinoid receptor agonists inhibit tumor growth and metastasis of breast cancer.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/19457575
    Cannabinoid-induced apoptosis in immune cells as a pathway to immunosuppression.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/16908594
    Delta9-tetrahydrocannabinol-induced apoptosis in Jurkat leukemia T cells is regulated by translocation of Bad to mitochondria.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/12130702
    Delta(9)-tetrahydrocannabinol-induced apoptosis in the thymus and spleen as a mechanism of immunosuppression in vitro and in vivo.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/11854771
    Cannabinoids and the immune system.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/20053780
    Cannabidiol enhances the inhibitory effects of delta9-tetrahydrocannabinol on human glioblastoma cell proliferation and survival.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/16754784
    Cannabidiol-induced apoptosis in human leukemia cells: A novel role of cannabidiol in the regulation of p22phox and Nox4 expression.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/20090845
    The expression level of CB1 and CB2 receptors determines their efficacy at inducing apoptosis in astrocytomas.

    ‪http://www.ncbi.nlm.nih.gov/pubmed/15978942
    Targeting cannabinoid receptors to treat leukemia: role of cross-talk between extrinsic and intrinsic pathways in Delta9-tetrahydrocannabinol (THC)-induced apoptosis of Jurkat cells.

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  17. Opa!!! Que bacana ver este video aqui no Hempadao, fui eu que editei. Na verdade o que fiz foi uma coletanea de mentiras e verdades sobre a erva retiradas de diversos videos do youtube. Fiz um video grande (Parte 1: www.youtube.com/watch?v=C0rssSbSFa8&feature=channel_video_title Parte 2: www.youtube.com/watch?v=HtBbG3a_reQ&feature=relmfu) e com este video fiz pequenos videos com os temas (Ex: maconha medicinal, maconha mata neuronios!? A maconha vicia...). Tem cenas do documentario cortina de fumaca, the union, penn and teller, debate da folha dentre outros... a ideia eh ajudar o trabalho feito por este site e outros que visam falir o traficante - legalizacao/descriminalizacao/regulamentacao da maconha! Vamos quebrar estes mitos ridiculos que fazem que uma planta benefica, que nunca matou ninguem, seja fonte de renda para o crime organizado!!! Abraco!! Eduardo, Curitiba, PR, Brasil (www.youtube.com/UrgentePelaPaz)

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  18. Ahhh, e a trilha eh toda Tom Ze!!!

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  19. to na espera do dia em q o hempadao poste cortina de fumaça...

    NAO CONSEGUIRÃO SUSTENTAR ESSA MENTIRA PRA SEMPRE!

    eu planto minha cannabis a menso de 1 mês e ja me sinto livre desse sistema, entre trafico e pulícia,. espero od ia em q eu póssa colher meus próprios camarões.

    Rafaeal w.

    alguém sabe de fertilizante naturaL q eu póssa usar?

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